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0:08
ACE-inhibitor–induced angioedema occurs due to bradykinin accumulation, as ACE normally degrades bradykinin. This leading to increased vascular permeability and sudden facial and/or airway swelling. Risk factors include prior angioedema, female sex, older age, smoking, chronic kidney disease, and concomitant use of painkillers. Management requires immediate discontinuation of the ACE inhibitor, careful airway monitoring, and supportive treatment. Antihistamines and steroids have limited benefit.
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This is a fantastic question about ACE inhibitors and one of the rare side-effects , angioedema. Thank you for asking it Teresa. Don’t for get to subscribe if you want your questions answered. | ECT4Health- Nurse/Paramedic Education
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Angioedema is not specific to the face, but ACE Inhibitor-related angioedema is. But...it's not a histamine mediated allergic reaction. So it won't respond to Benadryl and is unlikely to respond to Epinephrine. So what do we do? | EMS Avenger
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We explore the 4 critical issues linked to ACE inhibitors: angioedema, cough, hyperkalemia, & intrinsic renal disease. #ACEInhibitor #MedicationSideEffects #Angioedema #Hyperkalemia #RenalDisease #Pharmacology #Healthcare #MedicalEducation #KidneyHealth #DrugSafety #CME4Life #JohnBielinski #CME #PANCE #PANRE #PA #PhysicianAssistant #NP #ClinicalPearls #PANCEPearls #ClinicalPractice #StudySmarterNotHarder #TextPimp #ACEI #ACE | CME4LIFE
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